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| liver failure, glycogen storage disorder, alcoholic steatosis, varices, childhood onset diabetes, inflammation, chest pain, wheat grass, lipodystrophy, plump dj's , plump jack squaw valley , real plump , environment, white girls with fat asses , plump galleries , natural, plump asian , dr. mirkin, gallbladder disease, parrots, omega3 fatty acid , medical photograph, find information, cholesterol, | Underlying causes include obesity, corticosteroids, diabetes, malnutrition, 'reye's syndrome' total parenteral nutrition, alcohol abuse, chemotherapy, and toxin exposure such as carbon tetrachloride. It appears as focal, geographic, or diffuse areas of hypoattenuation. Liver parenchyma commonly spared from 'reye's syndrome' fatty infiltration is found around the portal vein and bifurcation, around the gallbladder fossa, near the falciform ligament, and in the quadrate lobe. Fatty infiltration does not disrupt the path of the normal liver vasculature that traverse through these areas of heterogeneity. The Budd-Chiari syndrome also demonstrates diffuse heterogeneity similar to passive venous congestion. However, disproportionate enlargement and 'reye's syndrome' enhancement of the caudate lobe are common features. Furthermore, the retrohepatic IVC is commonly narrowed secondary to the caudate lobe hypertrophy. |
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| Larger patchy areas of poor or delayed enhancement near the periphery are childhood onset diabetes probably a manifestation of nearly stagnant blood flow. This appearance may mask small underlying neoplastic lesions and biopsy may be necessary. If the underlying cause is corrected, CT abnormalities may resolve.1,3 Although cirrhosis can appear with diffuse heterogeneity as well, other childhood onset diabetes findings will most likely be present. Cirrhotic livers will also show either generalized hepatomegaly or specific hypertrophy of the caudate lobe with regression of the childhood onset diabetes right liver lobe. A nodular contour of the liver may also be seen. Focal inhomogeneities of the cirrhotic liver other than hepatomas may include fatty infiltration or areas of sparing or regenerating nodules. Associated findings of subsequent portal venous hypertension (eg, an enlarged portal vein, varices, splenomegaly, and ascites) also may be seen. Fatty infiltration of the liver can also appear resulting from triglyceride deposition within the hepatocytes. |
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