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plump rumps , lupus, fibroids of uterus, liver function, holistic, plump girl , inflammatory arthritis, simple steatosis, medicine, liver enzymes, big n plump , plump dumpling , sexy plump , online, | In the US: Steatosis affects approximately 25% of the general population. Steatohepatitis may be related to alcohol-induced hepatic damage or may be unrelated to alcohol (ie, NASH). NASH has been detected in 1.2-9% of patients undergoing routine liver biopsy. NAFLD is found in over 80% of patients who are obese. Over 50% of patients undergoing bariatric surgery have NASH. Mortality/Morbidity: Steatosis is a benign condition, fat bottomed girls and progression is very rare. Steatohepatitis may progress to liver fibrosis and cirrhosis and may result in liver-related fat bottomed girls morbidity and mortality. Fibrosis or fat bottomed girls cirrhosis in the liver is present in 15-50% of patients with NASH. Approximately 30% of patients with fibrosis develop cirrhosis after 10 years. Many cases of cryptogenic cirrhosis may represent so-called burnt-out NASH because a high proportion is associated with obesity, type II diabetes, or hyperlipidemia. |
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per week. Identical lesions also can be caused by other diseases or toxins. If steatohepatitis simple steatosis is present but a history of alcohol use is not, the condition is termed nonalcoholic steatohepatitis (NASH). Fatty change in the liver results from excessive accumulation of lipids within hepatocytes. Simple fatty liver is believed to be benign, but NASH can progress to cirrhosis and can be associated with hepatocellular carcinoma. These changes are recognized on liver biopsy specimens, and, thus, the diagnoses simple steatosis are morphological rather than clinical.Pathophysiology: Fatty liver is the accumulation of triglycerides and other fats in liver cells. In some patients, this may be accompanied by hepatic inflammation and liver cell death (steatohepatitis). Potential pathophysiological mechanisms include (1) decreased mitochondrial fatty acid beta-oxidation, (2) increased endogenous fatty acid synthesis or enhanced delivery of fatty acids to the liver, and (3) deficient incorporation or export of triglycerides as very-low density lipoprotein.Frequency: |
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