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| fatty infiltration of liver , hepatic steatosis, auto immune reaction, gabe mirkin, bobby plump , plump princess , merkin, ulcerative colitis, online submission, galactosemia, headaches migraines, | The intense enhancement on the arterial phase may be difficult to distinguish from hypervascular metastasis, especially if enhancement is heterogeneous (Figure 17). - Hepatic adenoma Hepatocellular adenoma is a rare benign lesion, colitis most often seen in young women, with a history of oral contraceptive use. Uncomplicated adenomas are typically solitary, sharply marginated and may contain fat. Small lesions enhance rapidly and colitis are hyperattenuating relative to the liver. Adenomas may be mistaken for malignant lesions (Figure 18) especially if they colitis are large, multiple or hemorrhagic. E) Indeterminate lesions When benign lesions have atypical features they may be considered indeterminate (Figures 19, 20). In our study 26 out of 237 lesions (11%) were considered indeterminate by at least 1 reviewer. The mean size of these lesions was 1.5 cm (range 0.2 |
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| They are usually subcapsular in location. Calcification (Figure 14) or vessels (Figure 15) in the wall of a lesion could result in misclassification of metastasis or other malignant lesions as hemangioma. - Atypical hemangioma If hemangiomas lack the classical features, they may be misclassified as malignant. Small lesions may fill completely in the arterial phase, and may be difficult to distinguish form hepatic steatosis hypervascular metastasis. When partially exophytic, hemangioma may lack the peripheral nodular enhancement, resulting in misdiagnosis (Figure 16). - Focal nodular hyperplasia Focal nodular hyperplasia are well defined lesions with intense homogeneous enhancement hepatic steatosis on hepatic steatosis the arterial phase images, that disappears on the portal venous phase. If a central scar is present it demonstrates delayed enhancement. |
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